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Publication facts (s) having Tepper JM, Tecuapetla F, Koós T, Ibáñez-Sandoval O (2010) as Paper having this fact


 
Name
1 D5 and D2 dopamine receptors induce "depolarization and an increase in input resistance in striatal FSI in brain slices"
2 FSIs “have GABAergic inputs originating from the GP. The pallidostriatal inputs are largely selective for FSIs (Bevan et al., 1998) and in vivo, increased firing of FSI during choice selection in a simple discrimination task coincide with a decrease in firing of GP neurons .”
3 Perhaps the principal function of these neurons is to release SOM, NOS, and/or NPY, all of which could exert slower neuromodulatory effects on their postsynaptic targets rather than fast synaptic effects. For example, SOM has been shown to exert a potent presynaptic inhibition on GABA release at SPN–SPN synapses.”
4 PLTS interneurons were found to evoke only sparse and relatively weak GABAergic IPSCs in SPNs.”
5 Reviewed in
6 Striatal FSIs make [GABAergic] synapses onto both direct and indirect path- way SPN. The biophysical properties of the synaptic contacts do not differ and exhibit short-term depression. Further, single FSI often make synapses with both types of SPN”
7 The most characteristic attributes of these neurons were the presence of a low threshold Ca2+ spike."
8 While nicotinic and muscarinic acetylcholine receptors exist in FSI-SPN synapses, only the nicotinic receptors are postsynaptic.