| 401 |
In mouse retinal bipolar cells, T-type Ca currents were recorded in soma, while L-type currents were recorded from axonal terminal |
| 402 |
In patch recordings, "HVA-l channels reminiscent of L-type channels were occasionally encountered primarily in the more proximal dendrites" (and in the soma) |
| 403 |
In PLTS cells “influx of Ca2+, while persistent depolarizations were due to influx of Na+. ” |
| 404 |
In primary culture, GABA and glycine exert inhibitory actions on olfactory bulb neurons, mitral/tufted cells, granule and periglomerular cells |
| 405 |
In retinal bipolar cells of bullfrog, both axon terminals and dendrites showed high GABA sensitivity mediated by both GABA(A) and GABA(C) receptors. GABA(A) and GABA(C) receptors may play different roles in the outer and inner retina and the differential complements of the two receptors on OFF and ON BCs may be closely related to physiological functions of these cells |
| 406 |
In Salamander |
| 407 |
In situ hybridization of three cloned SK channel subunits (SK1-3), the prime candidates likely to underlie Ca(2+)-dependent AHPs showed high levels of expression in regions presenting prominent AHP currents including CA1-3 regions of the hippocampus (SK1 and SK2), reticularis thalami (SK1 and SK2), supraoptic nucleus (SK3), and inferior olivary nucleus (SK2 and SK3) |
| 408 |
In situ hybridization of three cloned SK channel subunits (SK1-3), the prime candidates likely to underlie Ca(2+)-dependent AHPs showed high levels of expression in regions presenting prominent AHP currents including CA1-3 regions of the hippocampus (SK1 and SK2), reticularis thalami (SK1 and SK2), supraoptic nucleus (SK3), and inferior olivary nucleus (SK2 and SK3) |
| 409 |
In slices of rat brain at various postnatal ages was found that decay times of evoked IPSCs and spontaneous miniature IPSCs undergo progressive shortening during the first postnatal month |
| 410 |
In slices that preserve mossy-fiber to granule cell synapses, Ach induced diverse responses in granule cells, one response being somatic current (nAChR also mediated postsynaptic currents, PSCs, which, however, were glutamatergic in nature, indicating a presynaptic mechanism. ) |
| 411 |
in SOBIV 297. |
| 412 |
In the dorsolateral striatum, only parvalbumin mRNA-positive neurons expressed the mRNA encoding the potassium channel Kv3.1, a member of the Shaw family of potassium channels with rapid activation and inactivation kinetics, usually found in fast-firing neurons such as the basket cells of the hippocampus.” |
| 413 |
In the hair-cell microvilli |
| 414 |
In the hair-cell microvilli (Hudspeth AJ, Corey DP, others) |
| 415 |
In triads made up of (i) a cholinergic axon, (ii) one or several periglomerular or granule cell dendrites, and (iii) usually one relay cell dendrite, asymmetric cholinergic synapses were selectively focused on dendrites (gemmules and spines) of periglomerular or granule cells. |
| 416 |
In vitro, however, blocade of AMPA, NMDA, GABAA,D1 , D2, muscarinic receptors affect little the firing frequencies and patterns of cholinergic interneurons, indicating that these neurons are endogenously active and generate action potentials in the absence of any synaptic input |
| 417 |
In vivo, the excitatory influence of STN contributes to the tonic discharge of SNr cells as shown by the marked reduction in SNr firing rate induced by a pharmacological blockade of STN activity |
| 418 |
Inactivation of dendritic Na channel contributes to the attenuation of activity-dependent backpropagation of APs |
| 419 |
Increased conductance may follow Ca impulses |
| 420 |
Increased conductance may follow Ca impulses (Llinas and Walton 1998). |
| 421 |
indicating that Ia synapses are distributed widely over soma-dendrites (confirmed by HRP labelling of Ia afferents on labelled motoneurones: reviewed in |
| 422 |
Indirect evidence for this current was found in O-A interneurons |
| 423 |
Indirect experimental evidence for dendritic Na channels was suggested by laminar filed potential studies |
| 424 |
Individual branches can function as single integrative compartments where the fast oblique spike contains contributions from NMDA, VGCCs, and the A current |
| 425 |
inferred |
| 426 |
Inferred in |
| 427 |
Inhibition of deep neurons (pyramidal and multipolar) by (presumed?) deep layer interneurons involves a fast Chloride and a slow potassium-mediated IPSP |
| 428 |
Inhibitory interneurons, recorded mostly in the deeper part of layer II of piriform cortex, mediate fast IPSPs in principal cells and are activated at least partly through the axon collaterals of the principal cells |
| 429 |
Input from dopaminergic neurons in the substantia nigra; causes small depolarization? sometimes a decrease in firing rate; modulates anomalous rectification of dendritic membrane (I IR)? |
| 430 |
Intracellular recording from CA3 pyramidal neurons in a slice culture from rat found a slow excitatory response to Glu application in the presence of blocking agents for the ionotropic GluRs. Further experimentation revealed that ACPD could evoke the same response, which was due to the depression of I(K, Ca) and voltage-gated I(K) |
| 431 |
Intracellular recording from CA3 pyramidal neurons in a slice culture from rat found a slow excitatory response to Glu application in the presence of blocking agents for the ionotropic GluRs. Further experimentation revealed that ACPD could evoke the same response, which was due to the depression of I(K,Ca) and voltage-gated I(K) |
| 432 |
Intracellular recording from organotypic rat hippocampal cultures have shown that approximately 25% of single spikes in CA3 pyramidal neurons are followed by IPSPs at a fixed latency, presumeably a result of feedback inhibition from inhibitory interneurons. The addition of bicuculline (a competitive GABA(A) antagonist) completely abolished these responses, but they were insensitive to CGP35348, a GABA(B) antagonist |
| 433 |
Intracellular recordings from sensorimotor cortex suggested that what activate IKCa persistently would not be calcium but some biochemical modification triggered by NMDA receptor activation |
| 434 |
Intracellular recordings showed that Apamin-sensitive IKCa regulate pacemaker activity in these neurons |
| 435 |
Intracellular recordings suggested different functional consequences for modulation of Ca2+ current subtypes. Based on the effects of specific organic Ca2+ channel blockers the sAHP was found to be coupled to N-, P-, and Q-type currents. P-type currents were coupled to the mAHP |
| 436 |
Intracellular recordings: activation by I AHP causes unstable depolarizing plateau potentials; activated and modulated by 5-HT for decending raphe axons |
| 437 |
Intracellular recordings: afterhyperpolarization potential inverses during repetitive impulse firing, limiting durations and frequency range of impulse firing |
| 438 |
Intracellular recordings: AP5 blocks late component of EPSP elicited by olfactory nerve volley |
| 439 |
Intracellular recordings: AP5 blocks late component of EPSP response to olfactory nerve volley |
| 440 |
Intracellular recordings: AP5 blocks late component of EPSP response to olfactory nerve. volley |
