| 441 |
Intracellular recordings: AP5 blocks late component of EPSP response to olfactory nerve. volley |
| 442 |
Intracellular recordings: CMQX and NBQX applied at the soma completely block short duration (i.e. near soma) single fiber EPSPs |
| 443 |
Intracellular recordings: CNQX blocks early component of EPSP elicited by olfactory nerve volley |
| 444 |
Intracellular recordings: CNQX blocks early component of EPSP elicited by olfactory nerve volley nerve volley |
| 445 |
Intracellular recordings: CNQX blocks early component of EPSP response to olfactory nerve volley |
| 446 |
Intracellular recordings: IPSP blocked by bicuculline and low Cl- |
| 447 |
Intradendritic recordings show Ca-dependent plateau potentials and Ca impulses |
| 448 |
Intrastriatal stimulation-induced release of ACh from presynaptic nerves hyperpolarizes cholinergic interneurons by activating muscarinic receptors (probably M2) located on somatodendritic region |
| 449 |
Involvement of presynaptic NMDA receptors in cerebellar long-term depression at parallel fiber-Purkinje cell synapses |
| 450 |
Ionophoretic glutamate applied to dendrites depolarizes Purkinje cells. Glu is released from parallel fibers of granule cells |
| 451 |
IP3 gated ion channel; found in soma by |
| 452 |
IPSP blocked by bicuculline |
| 453 |
IPSP blocked by bicuculline and low Cl- |
| 454 |
IPSPs elicited by monoamines in pyramidal cells result from a convergence of inputs from populations of layer II/III interneurons that are activated by one, two or all three of the following monoamines 5-HT, NE, and DA. |
| 455 |
Isolated rod-dominant on-center bipolar cells respond to GABA, the highest sensitivity of which being located at the axon terminal |
| 456 |
It contributes to the spontaneous activity of these cells and to the tonic inhibition of CA1 pyramidal neurons in the hippocampus |
| 457 |
It has also been found that CNQX does not block the intracellular calcium concentration increase normally associated with stratum lucidum stimulation |
| 458 |
It has also been found that CNQX does not block the intracellular calcium concentration increase normally associated with stratum lucidum stimulation |
| 459 |
It has also been shown (using whole and perforated patch recording from acutely isolated CA3 pyramidal neurons) that application of Glu and quisqualatic acid (in the presence of D-AP5, an NMDAR-antagonist, and CNQX, an AMPAR antagonist) results in responses that consist of an inward current that may be preceded by an outward current. Both of these currents are affected by bath [K] and they had different pharmacological properties (Harata et al, 1996). |
| 460 |
It has also been shown (using whole cell and perforated patch recording from acutely isolated CA3 pyramidal neurons) that application of Glu and quisqualatic acid (in the presence of D-AP5, an NMDAR-antagonist, and CNQX, an AMPAR antagonist) results in responses that consist of an inward current that may be preceded by an outward current. Both of these currents are affected by bath [K] and they had different pharmacological properties (Harata et al. 1996 #8680866). |
| 461 |
It has been shown that activation of these receptors could facilitate transmitter release. Their activation is very fast (<10 ms) and lasts for seconds, and could contribute to the short-term plasticity characteristics of mossy fiber synapses |
| 462 |
it has been shown to occur at mossy fiber synapses even in the presence of NMDA receptor antagonists under certain conditions |
| 463 |
It has been suggested in rats that GABA(B) receptors modulate dendrodendritic inhibition primarily by inhibiting granule cell calcium channels and reducing the release of GABA |
| 464 |
It has been suggested that the pharmacologically separable components of the HVA current in these neurons do not differ significantly in kinetics |
| 465 |
It has been suggested that this current is not involved in the generation of AHP but (with other HVA currents) contributes to the inward currents that regulate interspike intervals during repetitive firing |
| 466 |
It has been suggested that voltage-gated calcium channels play a role in LTP |
| 467 |
It has been suggested that voltage-gated calcium channels play a role in LTP (Johnston et al. 1992), and it has been shown that nimodipine (an L-channel antagonist) prevents certain mossy fiber LTP-types from taking place |
| 468 |
It was decreased by cannabinoids |
| 469 |
It was shown that stimulation of PG cells results in self-inhibition: release of GABA from an individual PG cell activates GABA(A) receptors on the same neuron |
| 470 |
It was was blocked by linopirdine in a reversible, concentration-dependent manner |
| 471 |
Kinetic properties were studied using the whole-cell patch-clamp technique |
| 472 |
Kinetic, pharmacological, and structural properties of these receptors were studied using patch-clamp techniques and single-cell reverse transcriptase polymerase chain reaction |
| 473 |
Korf et al, 1976; Cheramy et al 1981) by backpropagating impulse |
| 474 |
L-type ICa was found only in cells that retained axon terminals ramifying in the inner plexiform layer |
| 475 |
Labeling for glutamic acid decarboxylase (GAD), the enzyme that synthesizes GABA, is heavy in the molecular layer of CA1 |
| 476 |
Large EPSPs activate I IR, increasing the membrane resistance, shortening the dendrites electrotonically, making the cell more sensitive to subsequent inputs (see |
| 477 |
Lateral circuits contribute to the inhibitory surround. The bipolar neurons release glycine or GABA onto presynaptic bipolar neurons and ganglion cell dendrites |
| 478 |
Layer 2/3 fast-spiking interneurons (most of which are basket cells) received the strongest excitatory input (presumably glutamatergic) from layer 4 |
| 479 |
Layer 4 basket cells in cat visual cortex receive asymmetric synapses from layer 6 pyramidal (~43%), the spiny stellate (44%) and thalamic afferents (13%) |
| 480 |
Layer 4 basket cells in cat visual cortex receive excitatory input from neighboring spiny neurons, resulting in large excitatory EPSPs. One third of spiny cell-smooth cell pair were connected reciprocally (postsynaptic targets include local spiny stellate, pyramidal cells, as well as smooth neurons), but the postsynaptic IPSPs evoked by basket cells are slower, GABAergic, probably of GABAA type |
