| 201 |
by contrast, |
| 202 |
By using dendritic recordings and calcium transients in rats it was shown that this current may control AP propagation in lateral dendrites |
| 203 |
Ca -dependent Chloride current at the presynaptic terminals of goldfish retinal bipolar cells |
| 204 |
Ca fluorescence imaging shows that application of L-channel antagonists reduces the Ca influx associated with backpropagating action potentials, and has a significantly greater effect in the proximal dendrites than in more distal dendrites |
| 205 |
Ca fluorescence imaging shows that application of N-channel antagonists slightly reduces the Ca influx associated with backpropagating action potentials |
| 206 |
Ca fluorescence imaging shows that application of P-channel antagonists reduces the Ca influx associated with backpropagating action potentials |
| 207 |
Ca-dependent potassium currents are seen in dissociated cells |
| 208 |
CA1 neurons and subiculum neurons in hippoampus differ in firing pattern (the former being regular and the later being either regular, weakly bursting or strongly bursting) and resting membrane properties (such as input restistance and membran time constant); however, low concentration of 4-AP (50 µM) can convert neurons in both regions into firing bursting action potentials |
| 209 |
CA1 neurons and subiculum neurons in hippoampus differ in firing pattern (the former being regular and the later being either regular, weakly bursting or strongly bursting) and resting membrane properties (such as input restistance and membran time constant); however, low concentration of 4-AP (50 ?M) can convert neurons in both regions into firing bursting action potentials |
| 210 |
CA1 neurons and subiculum neurons in hippoampus differ in firing pattern (the former being regular and the later being either regular, weakly bursting or strongly bursting) and resting membrane properties (such as input restistance and membran time constant); however, low concentration of 4-AP (50 µM) can convert neurons in both regions into firing bursting action potentials |
| 211 |
CA1 pyramidal neurons increase their firing (recorded extracellularly) in response to ionophoresed Glu within their apical dendritic fields or in the cell body layer (Dudar 1974 PMID#4437726). |
| 212 |
Ca2+ -activated K+ current at presynaptic terminals of goldfish retinal bipolar cells |
| 213 |
Ca2+-activated K+ currents were studied using whole-cell patch-clamp recordings from freshly dissociated mouse neocortical pyramidal neurons |
| 214 |
Calcium entry through N-type calcium channels (CaV2.2) causes activation of KCa channels that decrease the firing rate and increase the regularity of firing in SNr GABA neurons” |
| 215 |
Calcium influx through NMDA receptors can directly trigger presynatic GABA release for local dendrodendritic feedback inhibition. DDI is elicited by photorelease of caged Ca++, with and without Cd++ and Ni++. |
| 216 |
Calcium influx through NMDA receptors directly evokes GABA release in granule cells. |
| 217 |
Calcium is involved in delayed release of neurotransmitter at synapses between granule cell their postsynaptic targets (stellate cells and Purkinje cells) |
| 218 |
Calcium-dependent potassium channels are preferentially activated by calcium entry through N- and Q-type channels |
| 219 |
CaV channels are among the intrinsic membrane channels influencing the excitability of the SNr neuronal membrane. CaV channels have been shown to influence spontaneous activity in these neurons, as well as more complex behaviors such as plateau potential generation burst firing.” |
| 220 |
Cell-attached dendritic recordings in rats up to about 60um from the soma showed that low-threshold calcium channels were concentrated at proximal dendritic locations, sites known to receive excitatory synaptic connections from primary afferents, suggesting that they play a key role in the amplification of sensory inputs to TC neurons |
| 221 |
Cell-attached patches on the proximal 100um of the apical dendrite did not contain sAHP channels. Amputation of the apical dendrite approximately 30 micron from the soma, while simultaneously recording the sAHP whole cell current at the soma, depressed the sAHP amplitude by only approximately 30% compared with control. Somatic cell-attached and nucleated patches did not contain sAHP current. Amputation of the axon about 20um from the soma had little effect on the amplitude of the sAHP. By this process of elimination, it is suggested that sAHP channels may be concentrated in the basal dendrites of CA1 pyramids |
| 222 |
Cell-attached recordings in rats up to about 60um from the soma demonstrated a non-uniform dendritic distribution of channels |
| 223 |
Cell-attached recordings in rats up to about 60um from the soma demonstrated a roughly uniform density of channels across the somatodendritic area examined that corresponds to approximately half the average path length of TC neuron dendrites |
| 224 |
Cell-specifc modulation of GABAA receptor- mediated chloride current by dopamine. In interneurons (mainly granule cells), dopamine reduces GABAA Cl- current, via D1 receptor and involves phosphorylation of GABAA receptors by PKA. In mitral cell, dopamine enhances GABA responses via activation of D2 receptors and phosphorylation of GABAA receptors via PKC. |
| 225 |
Cell-specifc modulation of GABAA receptor- mediated chloride current by dopamine. In interneurons (mainly granule cells), dopamine reduces GABAA Cl- current, via D1 receptor and involves phosphorylation of GABAA receptors by PKA. In mitral cell, dopamine enhances GABA responses via activation of D2 receptors and phosphorylation of GABAA receptors via PKC. |
| 226 |
Cells acutely dissociated from slices obtained from chronic temporal lobe epilepsy patients displayed a high-voltage activated Ca2+ conductance with a pronounced Ca2+-dependent inactivation |
| 227 |
Cells were voltage-clamped using a single microelectrode, at 23-30 degrees C. M-current resembled that of sympathetic ganglion cells. It was abolished by addition of carbachol, muscarine or bethanechol, as well as by 1 mM barium. It was suggested that activation of cholinergic septal inputs to the hippocampus facilitates repetitive firing of pyramidal cells by turning off the M-conductance, without much change in the resting potential of the cell |
| 228 |
Cerebellar granule cells from young rats (postnatal days 1-9) possess voltage-activated inward Na+ current as well as two types of K+ current, IA and IK |
| 229 |
Chen et al 1997). Dendritic patch recordings showed an even density of Na channels (120pS um-2) up to 350 um from the soma along the primary dendrite to the origin of the glomerular tuft |
| 230 |
Chen et al 1997).Dendritic patch recordings showed an even density of Na channels (120pSum-2) up to 350 um from the soma along the primary dendrite to theorigin of the glomerular tuft |
| 231 |
Choline acetyltransferase immunocytochemistry shows that cholinergic projections from the basal forebrain to the main olfactory bulb focus synaptic innervation on interneurons, on the dendritic spines of periglomerular and granule cells. |
| 232 |
Cholinergic agonist carbachol caused a significant suppression of inhibitory postsynaptic potentials (IPSPs) in the pyramidal neurons that were induced by stimulation of layer Ib, with a weaker effect on IPSPs induced by stimulation of layer Ia |
| 233 |
Cholinergic agonist carbachol selectively suppresses intrinsic fiber synaptic potentials but not afferent fiber synaptic potentials. |
| 234 |
Cholinergic agonist carbachol selectively suppresses intrinsic fiber synaptic potentials but not afferent fiber synaptic potentials. A presynaptic mechanism of cholinergic suppression is suggested |
| 235 |
Cholinergic agonist carbachol selectively suppresses intrinsic fiber synaptic potentials but not afferent fiber synaptic potentials. (Hasselmo ME and Bower JM, 1992353 ) (Patil MM and Hasselmo ME, 1999341 ). |
| 236 |
Cholinergic interneurones in rat exhibit a ATP-sensitive potassium channel current. sigle-cell-RT-PCR shows that this channel is formed from Kir6.1 and SUR1 subunits |
| 237 |
cited in Johnston and Amaral, 1998). |
| 238 |
Clusters of glycine receptors were found on the somatodendritic membranes of Alpha ganglion cells |
| 239 |
Clusters of the alpha1, and alpha2, alpha3, and gamma2 subunits of the GABAA receptor were found on the somatodendritic membranes of Alpha ganglion cells. Experiments with different combinations of the subunit-specific antibodies showed that the alpha1, alpha2, and alpha3 subunits of the GABA(A) receptor are not colocalized within the same clusters, suggesting that an individual neuron can express several isoforms of the GABAA receptor and that these different isoforms are aggregated at distinct postsynaptic sites |
| 240 |
Combining preembedding and postembedding immunostaining at the EM level, GluR2/3 and NMDAR1 immunoreactivity was located in somata and in proximal and distal dendrites |
