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Publication facts - List


 
Name
241 Conduction of the action potential suggests there must be some Na channels there.
242 Cultured cells
243 Cultured cells; blocked by 100 uM Cd or nifedipine (IC50=368 nM); no T-type Ca channels found
244 Cultured cells; TTX-sensitive
245 Curare blocks responses to applied ACh.
246 D2 dopamine receptors reduce N-type Ca2+ currents in rat neostriatal cholinergic interneurons
247 D5 and D2 dopamine receptors induce "depolarization and an increase in input resistance in striatal FSI in brain slices"
248 DA is released from dendrites
249 DA receptors in mitral cell dendrites implied by DA localization in PG dendrites presynaptic to mitral dendrites
250 DDI (dendrodendritic inhibition) can be elicited by activation of AMPA receptors, while NMDA receptor activation is not an absolute requirement. DDI is blocked by Cd and toxins to N- and P/Q-type channels.
251 DDI (dendrodendritic inhibition) can be elicited by activation of AMPA receptors, while NMDA receptor activation is not an absolute requirement. DDI is blocked by Cd and toxins to N- and P/Q-type channels.
252 Deep multipolar cells have GABA-containing terminals arranged in baskets around pyramidal cell bodies
253 Delayed firing of action potentials in response to current pulse injection suggests there is I A current here. Unpublished data have characterized the kinetics of A current in the rat mitral cell
254 Delayed rectifier. (See SOBiv p140).
255 Dendritic can fire sodium spikes that can precede somatic action potentials (APs), the probability and amplitude of which depend on previous synaptic and firing history. Some dendritic spikes could occur in the absense of somatic APs, indicating that their propagation to soma is unreliable
256 Dendritic fluorescence imaging showed that Ca2+ channels of several subtypes mediated the AP-evoked fluorescence transient in the proximal (100-170 microns) apical dendrite. The fluorescence resulted from Ca2+ entry through L, N, and P-type channels, and through Ca2+ channels (R-type) not sensitive to L-, N- and P-type Ca2+ channel blockers
257 Dendritic GABAA-mediated IPSPs act largely independent of somatic IPSPs and may regulate facilitation of MNDA-mediated respnses
258 Dendritic patch recordings showed an even density of Na channels (120pS um-2) up to 350 um from the soma along the primary dendrite to the origin of the glomerular tuft
259 Dendritic patch recordings showed an even density of Na channels (120pSum-2) up to 350 um from the soma along the primary dendrite to theorigin of the glomerular tuft
260 Dendrodendritic from granule cell spines. IPSP blocked by bicuculline and low Cl-
261 Dendrodendritic inhibition (DDI) between mitral and granule cells relies on N-and P/Q- type calcium channels. Magnitude of DDI is proportional to dendritic calcium influx.
262 Dendrodendritic inhibition (DDI) between mitral and granule cells relies on N-and P/Q- type calcium channels. Magnitude of DDI is proportional to dendritic calcium influx.
263 Dendrodendritic inhibition between mitral and granule cells involves N-and P/Q- type calcium channels. The magnitude of DDI is proportional to dendritic calcium influx.
264 Dendrodendritic inhibition between mitral and granule cells involves N-and P/Q- type calcium channels. The magnitude of DDI is proportional to dendritic calcium influx.
265 Dendrodendritic synapse onto mitral/tufted cells, of type 2
266 Densities and kinetics were measured from patch clamp recordings
267 Depolarisations beyond -40 mV activated a fast transient TTX-sensitive inward current. Once activated, INa declined exponentially to zero following a single exponential. The underlying conductance showed a sigmoidal activation between -40 and +30 mV, with half activation at -17.4 mV and a maximal value of 9.7 nS per neurone. The steady-state inactivation was complete at -30 mV and completely removed at -90 mV, with a midpoint at -56 mV. The activation process could be adequately described by third order kinetics, with time constants ranging from 260 microseconds at -20 mV to 70 microseconds at +50 mV.
268 Depolarization induced transient outward currents that resembled IPSCs and were blocked by GABA and glycine receptor antagonists, suggesting that they arise from activation of amacrine feedback synapses
269 Depolarizes SNr principle cells. Generated by subset of voltage-gated sodium channels that remain open for extended periods of time. Can contribute to neuronal excitability and pacemaking activities.
270 Depolarizing "sag" during larger hyperpolarizing voltage transients is indicative of Ih current in determinating the passive membrane properties of CA1 pyramidal neurons
271 Different GABAergic receptors are localized to specific layers, and may mediate feedforward and feedback inhibitions
272 Differential GABABergic presynaptic modulation in layers Ia and Ib: While baclofen suppresses field potentials at the intrinsic fiber synapses proximal to the pyramidal cell bodies (layer Ib), it does not affect field potentials at distal dendrites (layer Ia)
273 Differential induction of potentiation and depression at commissural and mossy fiber synapses has also been shown by
274 Differential induction of potentiation and depression at commissural and mossy fiber synapses has also been shown by #R#158#E#. Recordings from membrane patches of dendrites and soma reveal fast and slow responses to fast application of glutamate, mediated by AMPA amd NMDA receptors, respectively
275 discussed in Burke 1998).
276 Dopamine modulates Ih in cultured rat olfactory receptor neurons
277 Dopaminergic afferents from substantia nigra pars compacta” target the globus pallidus.
278 Dopaminergic subdivision of the periglomerular interneurons throughout classes of vertebrates.
279 Dual patch recordings and Ca2+ imaging of mitral cells in the rat olfactory bulb slice suggested that action potentials propagating into the basal dendrites decrement approximately 20% per 100um
280 Dual patch recordings and Ca2+ imaging of mitral cells in the ratolfactory bulb slice suggested that action potentials propagating intothe basal dendrites decrement approximately 20% per 100um