Name
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521	NMDA receptor is required for DDI (dendrodendritic inhibition) since IPSC was completely blocked by AP-5. Ineffectiveness of AMPA receptor-mediated EPSPs to activate the granule cells may be due to their intrinsic membrane properties.
522	NMDA receptors play a critical role in dendrodendritic inhibition between mitral and granule cells. Moreover, N- and P/Q type calcium channels are involved.
523	NMDA reversibly depresses postsynaptic currents, through a trans-synaptic mechanism that involves release from parallel fibers nitric oxide that decreases the glutamate sensitivity of the Purkinje cell
524	Nomarski optics and infrared videomicroscopy were used to demonstrate the existence of a TTX-sensitive persistent Na+ conductance (INaP) in identified medium-sized neostriatal neurons
525	Nucleated patches revealed a fast component highly sensitive to external 4-AP and TEA (~57% of total K+ current) and a slow component that was sensitive to high concentrations of TEA, but insensitive to 4-AP (~25% of total K+ current)
526	Numerous authors (e.g.,
527	ON beta Ganglion cells. ON beta Ganglion cells respond to ionophoresed GLU and GLU agonists, and are blocked by GLU antagonists (SOBiv p238). Ganglion cells express GLUR and NMDAR
528	on spines (SOBiv p126,130). The neurotransmitter is GLU
529	One STN regulator is the serotonin (5-HT) system. The STN receives a dense 5-HT innervation. 5-HT1A, 5-HT1B, 5-HT2C, and 5-HT4 receptors are expressed in the STN. 5-HT may regulate the STN via several mechanisms."
530	Original intracellular recordings in vivo
531	P)
532	P2
533	p395).
534	p396
535	p396).
536	p396.
537	p397).
538	p397.
539	Paired recordings in slices showed excitatory transmission mediated solely by transmitter spillover between mitral cells. Dendritic glutamate release causes self-excitation via local activation of NMDA receptors, and generates NMDA receptor-mediated responses in neighbouring cells. It is suggested that this simultaneous activation of neighbouring cells by a diffuse action of glutamate provides a mechanism for synchronizing olfactory principal cells
540	Paired whole-cell recording revealed reciprocal excitatory connections between mitral cells. Pharmacological analysis suggested that it could be mediated by both AMPA and NMDA receptors
541	Patch Clamp recordings revealed density levels are similar to that found in the soma, with slightly different kinetics
542	Patch recordings
543	Patch recordings indicate channels similar in basic characteristics to one or more of the HVAm channel types (most likely Q- or R-type channels)
544	Patch recordings of back propagating impulses in dendrites. Variable densities of active channels support variable extents of backpropagating impulse in the dendrites
545	Patch recordings yield an approximate channel density of 28 pS/micron^2 in juvenile rats < 4 wks of age, rising to 61 pS/micron^2 in older rats. Channel density was similar in other dendritic compartments
546	Patch recordings yield an approximate channel density of 45 pS/micron^2 (compared with 28 pS/micron^2 in dendrites) in juvenile rats < 4 wks of age, rising modestly to 56 pS/micron^2 (compared with 61 pS/micron^2 in dendrites) in older rats
547	Patch recordings yield an approximate channel density of 7 pS/micron^2 in juvenile rats < 4 wks of age, rising to 10 pS/micron^2 in older rats. Ca channel density was similar in other dendritic compartments, and in general lower than Na channel density
548	Patch recordings yield an approximate channel density of 7 pS/micron^2 in juvenile rats < 4 wks of age, rising to 10 pS/micron^2 in older rats. Ca channel density was similar in other dendritic compartments, and in general lower than Na channel density
549	Patch-clamp recordings from human cells showed N-type, L-type and T-type currents that had similar pharmacological and kinetic characteristics as in control rats. The current density was significantly larger in human and in the kainate model compared to cells isolated from adult control rats
550	Patch-clamp recordings reveal a high density of A-type K channels in the dendritic tree, which increases with distance from the soma
551	Patch-clamp recordings reveal A-type K channels in the soma
552	Patch-pipette recordings found no evidence for a ?sag? in hyperpolarizing responses, suggesting that this current is not present in these neurons
553	Perforated whole-cell voltage-clamp recordings showed that dopamine modulates the L-type Ca2+ channels in rat olfactory receptor neurons via a voltage-independent mechanism
554	Perhaps the principal function of these neurons is to release SOM, NOS, and/or NPY, all of which could exert slower neuromodulatory effects on their postsynaptic targets rather than fast synaptic effects. For example, SOM has been shown to exert a potent presynaptic inhibition on GABA release at SPN–SPN synapses.”
555	Perhaps the principal function of these neurons is to release SOM, NOS, and/or NPY, all of which could exert slower neuromodulatory effects on their postsynaptic targets rather than fast synaptic effects. For example, SOM has been shown to exert a potent presynaptic inhibition on GABA release at SPN–SPN synapses.”
556	Periglomerular cells respond to microapplication of GABA, acetylcholine, norepinephrine and glycine with the activation of distinct ionic currents.
557	PG cells closely resembled previously described periglomerular cells in their morphology. During current clamp recording these neurons were characterized by their lack of action potentials upon depolarization. Consistent with these results no Na+ currents could be elicited in voltage clamp experiments. Two types of outward K+ currents were distinguished: one which inactivated and one which did not.
558	PKC may have a negative feedback role in modulating excitation by 5-HT in piriform cortical interneurons
559	PLTS cells exhibited unique firing properties due to Ca*+-dependent low-threshold spikes and Na+-dependent persistent depolarized spikes, in addition to Na+-dependent fast spikes.”
560	PLTS interneurons receive numerous synaptic contacts on their proximal dendrites from both cholinergic and dopaminergic axons, as well as onto their distal dendrites, which receive asymmetric synaptic inputs from the cortex.”