| 561 |
PLTS interneurons receive numerous synaptic contacts on their proximal dendrites from both cholinergic and dopaminergic axons, as well as onto their distal dendrites, which receive asymmetric synaptic inputs from the cortex.” |
| 562 |
PLTS interneurons were found to evoke only sparse and relatively weak GABAergic IPSCs in SPNs.” |
| 563 |
Possible |
| 564 |
Potassium-induced glutamate release from granule cells is dependent on the entry of Ca++ through multiple types of Ca-channel including N-, L-, P/Q-types; a number of these channel subtypes seems to be involved in the presynaptic modulation, by GABAB receptors, of potassium-induced glutamate release |
| 565 |
Potentiation of intrinsic excitability was induced by relatively weaker inputs than those that induce potentiation of synaptic efficacy; NMDA receptors are involved in both aspects of potentiation |
| 566 |
pp385,389,393-395,407). |
| 567 |
Presence of Kv4.2 but not Kv1.4 subunits in the somatodendritic membrane. Depolarization-activated potassium currents in cholinergic interneurons were dominated by a rapidly inactivating, K+-selective A current that became active at subthreshold potentials. |
| 568 |
Presence of Kv4.2 but not Kv1.4 subunits in the somatodendritic membrane. Depolarization-activated potassium currents in cholinergic interneurons were dominated by a rapidly inactivating, K+-selective A current that became active at subthreshold potentials |
| 569 |
Presence of Kv4.2 but not Kv1.4 subunits in the somatodendritic membrane. Depolarization-activated potassium currents in cholinergic interneurons were dominated by a rapidly inactivating, K+-selective A current that became active at subthreshold potentials |
| 570 |
Presence of Subthreshold-operating transient (A-type) K(+) currents (I(SA)s), as indicated by an accesspry subunit that accelerates the kinetics of this current |
| 571 |
Present in lower density than in the cilia |
| 572 |
Present in lower density than in the cilia.Properties of this channel in rat, and its functional interplay with the CNG channel, were studied by using inside-out membrane patches excised from ORN dendritic knobs/cilia |
| 573 |
presumably GABA inhibition (summarized in Young and Oertel, |
| 574 |
presumed |
| 575 |
Presynaptic terminal and axon NMDAR's have been found withimmunocytochemical techniques |
| 576 |
Probably. |
| 577 |
Prolongation of ISI through a D1 dopamine receptor-mediated enhancement of the afterhyperpolarization (AHP) |
| 578 |
Properties of K+ outward currents were investigated in human DG cells from 11 specimens obtained from patients with temporal lobe epilepsy. An IK was observed in all cells. The average current density, the time-dependent decay, and the resting membrane characteristics were not significantly different between patients with and without Ammon Horn Sclerosis. The V1/2(inact) was shifted in a hyperpolarizing direction in AHS (-67.7mV) compared with that in hippocampi not showing AHS (-47.7mV) |
| 579 |
Properties of potassium outward currents were investigated from 11 specimens obtained from patients with temporal lobe epilepsy. An IK but not IA or inwardly rectifying potassium currents, were observed in all cells |
| 580 |
Properties of this channel in rat, and its functional interplay with the CNG channel, were studied by using inside-out membrane patches excised from ORN dendritic knobs/cilia |
| 581 |
Properties of this current and its modulation by PKA were studied using whole-cell patch-clamp recording techniques |
| 582 |
provide evidence that activation of these receptors is necessary for LTP induction |
| 583 |
provide evidence that activation of these receptors is necessary for LTP induction . |
| 584 |
Quantitative autoradiography has been used to localize [3H]AMPA binding sites. In CA3, labeling was substantially heavier in s. pyramidale than in s.radiatum and s. lacunosum-moleculare |
| 585 |
Quantitative autoradiography has been used to localize [3H]AMPA binding sites. It was found that AMPARs are found in a high concentration in the hippocampus relative to other areas in the brain. In CA3, labeling was substantially heavier in s. pyramidale than in s.radiatum and s. lacunosum-moleculare |
| 586 |
Quantitative autoradiography has been used to localize sites at which L-[3H]-glutamate is displaced by NMDA. The labelling of these receptors was somewhat lower than in CA1 overall, being highest in s. oriens and s. radiatum and very low in s.pyramidale and s. lucidum |
| 587 |
quote from the review in Llinas and Walton 1990). |
| 588 |
Rat |
| 589 |
Re: PG cells: Two types of outward K+ currents were distinguished: one which inactivated and one which did not. |
| 590 |
Recording from dissociated neurons using intracellular and whole-cell voltage-clamp recordings showed that carbachol can act at M1-like muscarinic receptors to reduce the membrane K+ conductances and excite the neostriatal neurons |
| 591 |
Recordings from acute brain slices and in anesthetized rats using whole-cell recordings and Ca2+ imaging found that backpropagation of action potentials into the dendritic arbor is actively supported by Na+ channels both in vitro and in vivo. |
| 592 |
Recordings from acute brain slices and in anesthetized rats using whole-cell recordings and Ca2+ imaging found that single action potentials evoke little or none Ca2+ influx in the dendritic tuft, unless is paired with synaptic input |
| 593 |
Recordings from acute brain slices and in anesthetized rats using whole-cell recordings and Ca2+ imaging found that single action potentials evoke substantial Ca2+ influx in the apical trunk. |
| 594 |
Recordings from membrane patches of dendrites and soma reveal fast and slow responses to fast application of glutamate, mediated by AMPA amd NMDA receptors, respectively |
| 595 |
Recordings from membrane patches of dendrites and soma reveal fast and slow responses to fast application of glutamate, mediated by AMPA and NMDA receptors, respectively |
| 596 |
Recordings in slices showed that D1 receptor activation can either inhibit or enhance evoked activity, depending on the level of membrane depolarization, by modulating an L-type Ca2+ conductance |
| 597 |
Recordings in slices showed that GABA inhibition was mediated by GABA(B) receptors in the dendrites and GABA(A) receptors in the soma and dendrites |
| 598 |
Recordings using infrared-guided laser stimulation combined with whole cell recordings revealed a highly nonuniform distribution. Hot spots, with amplitude and integral of glutamate-evoked responses three times larger than responses evoked at neighboring sites, were detected. It appeared that the larger responses evoked resulted from an increase in activation of both AMPA and NMDA receptors. There was no correlation with branch points |
| 599 |
Recordings using the intracellular perfusion method showed no differences between the I-V characteristics of CA1 and CA3 neurones for this current. In contrast to this, the steady-state inactivation of both types of neurones was significantly different |
| 600 |
report the presence and function of Ih. |
