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Potassium-induced glutamate release from granule cells is dependent on the entry of Ca++ through multiple types of Ca-channel including N-, L-, P/Q-types; a number of these channel subtypes seems to be involved in the presynaptic modulation, by GABAB receptors, of potassium-induced glutamate release Show
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Activation of the adenosine A1 receptor reduces synaptic strength by modulating presynaptic calcium channels. Baclofen modulates presynaptic calcium channels as well but also affects release processes downstream from calcium entry Show
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Calcium is involved in delayed release of neurotransmitter at synapses between granule cell their postsynaptic targets (stellate cells and Purkinje cells) Show
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